Hip Dysplasia In Dogs

Hip Dysplasia in Dogs

Hip dysplasia is a hereditary disease that, in its more severe form, can eventually cause crippling lameness and painful arthritis of the joints. It is caused by a combination of genetic and environmental factors. It can be found in many animals and occasionally in humans, but is most commonly associated with dogs, and is common in many dog breeds, particularly the larger breeds.

Hip dysplasia is one of the most studied veterinary conditions in dogs, and the most common single cause of arthritis of the hips. [1] In humans it occurs at a rate of about 1 births per thousand (0.1%)

Overview

Normal hip anatomy

In the normal anatomy of the hip joint, the femur (the thigh bone) is connected to the pelvis at the hip joint. The almost spherical end of the femur head (the caput, or caput ossis femoris) fits into the acetabulum (a concave socket located in the pelvis). The bony surface of the femur head and of the acetabulum are covered by cartilage. While bones provide the strength necessary to support body weight, cartilage ensures a smooth fit and a wide range of motion. Normal hip function can be affected by congenital conditions such as dysplasia, discussed in this article, trauma, and by acquired diseases such as osteoarthritis and rheumatoid arthritis.

Dysplastic hip anatomy

In a hip suffering from dysplasia, two things are commonly abnormal. First, the caput is not deeply and tightly held by the acetabulum. Instead of being a snug fit, it is a loose fit, or a partial fit. Secondly, the caput or acetabulum are not smooth and round, but are misshapen, causing abnormal wear and tear or friction within the joint as it moves.

The body reacts to this in several ways. First, the joint itself is continually repairing itself and laying down new cartilage. However cartilage repair is a relatively slow process (the most rapid bodily repairs are often in systems with a blood flow, which cartilage lacks).

So the joint may suffer degradation due to the abnormal wear and tear, or may not support the body weight as intended. The joint becomes inflamed and a cycle of cartilage damage, inflammation and pain commences. This is a self-fueling process, in that the more the joint becomes damaged, the less able it is to resist further damage. The inflammation causes further damage. The bones of the joint may also develop osteoarthritis, visible on an X-ray as small outcrops of bone, which further degrade the joint.

The underlying deformity of the joint may get worse over time, or may remain static. A dog may have good X-rays and yet be in pain, or may have very poor X-rays and apparently almost no problems. The hip condition is only one factor to determine the extent to which dysplasia is causing pain or affecting the quality of life. In mild to moderate dysplasia it is often the secondary effects of abnormal wear and tear or arthritis, rather than dysplasia itself, which is the direct causes of visible problems.

Causes and effects

In dogs, there is considerable evidence that genetics plays a large role in the development of this defect. There might be several contributing genetic factors, including a femur that does not fit correctly into the pelvic socket, or poorly developed muscles in the pelvic area. Large and giant breeds are susceptible to hip dysplasia, and Cocker spaniels and Shetland sheepdogs are also known to suffer from it. Cats are also known to have this condition, especially Siamese.[1]

To reduce pain, the animal or person will typically reduce their movement of that hip. In animals this may be visible as "bunny hopping", where both legs move together, or less dynamic movement (running, jumping), or stiffness. Since the hip cannot move fully, the body compensates by adapting its use of the spine, often causing spinal, stifle (a dog's knee joint), or soft tissue problems to arise.

In dogs, the problem almost always appears by the time the dog is 18 months old. The defect can be anywhere from mild to severely crippling. It can cause severe osteoarthritis eventually.

It is most common in medium-large pure bred dogs, such as German Shepherd Dogs, Labrador or Golden retrievers, Rottweilers and Mastiffs, but also occurs in some smaller breeds such as spaniels.